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Epigenetic alteration of p16INK4a gene in dedifferentiation of liposarcoma.Academic Article Why?
Genes, p16Concept Why?
(-)-Epigallocatechin-3-gallate reactivates silenced tumor suppressor genes, Cip1/p21 and p16INK4a, by reducing DNA methylation and increasing histones acetylation in human skin cancer cells.Academic Article Why?
Sabaawy, HatimPerson Why?
Ectopic expression of the CDK inhibitor p21Cip1 enhances deregulated E2F activity and increases cancer cell-specific cytotoxic gene expression mediated by the ARF tumor suppressor promoter.Academic Article Why?
Radiation-induced gliomas represent H3-/IDH-wild type pediatric gliomas with recurrent PDGFRA amplification and loss of CDKN2A/B.Academic Article Why?
Acquisition of p16(INK4A) and p15(INK4B) gene abnormalities between initial diagnosis and relapse in children with acute lymphoblastic leukemia.Academic Article Why?
Multiplicity of abnormal promoter methylation in lung adenocarcinomas from smokers and never smokers.Academic Article Why?
p16 Loss and E2F/cell cycle deregulation in infant posterior fossa ependymoma.Academic Article Why?
The effect of flavopiridol on the growth of p16+ and p16- melanoma cell lines.Academic Article Why?
The inherent instability of mutant p53 is alleviated by Mdm2 or p16INK4a loss.Academic Article Why?
Glioma progression is shaped by genetic evolution and microenvironment interactions.Academic Article Why?
Robinson, WilliamPerson Why?
Aberrant DNA hypermethylation patterns lead to transcriptional silencing of tumor suppressor genes in UVB-exposed skin and UVB-induced skin tumors of mice.Academic Article Why?
Maloney, KellyPerson Why?
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